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Rosie D's List: Nutrition and Research

  • Jan 28, 10

    "Scientists have proved for the first time that fructose, a cheap form of sugar used in thousands of food products and soft drinks, can damage human metabolism and is fueling the obesity crisis.\n\nFructose, a sweetener usually derived from corn, can cause dangerous growths of fat cells around vital organs and is able to trigger the early stages of diabetes and heart disease.\n\nOver 10 weeks, 16 volunteers on a controlled diet including high levels of fructose produced new fat cells around their heart, liver and other digestive organs. They also showed signs of food-processing abnormalities linked to diabetes and heart disease. Another group of volunteers on the same diet, but with glucose sugar replacing fructose, did not have these problems."

    articles.mercola.com/...p-Alters-Human-Metabolism.aspx fructose sugar sweetener health

  • Jan 28, 10

    Consuming fructose-sweetened, not glucose-sweetened, beverages increases visceral adiposity and lipids and decreases insulin sensitivity in overweight/obese humans.\nStanhope KL, Schwarz JM, Keim NL, Griffen SC, Bremer AA, Graham JL, Hatcher B, Cox CL, Dyachenko A, Zhang W, McGahan JP, Seibert A, Krauss RM, Chiu S, Schaefer EJ, Ai M, Otokozawa S, Nakajima K, Nakano T, Beysen C, Hellerstein MK, Berglund L, Havel PJ.\nJ Clin Invest. 2009 May;119(5):1322-34. Epub 2009 Apr 20.\nPMID: 19381015\ndoi: 10.1172/JCI37385.\n\nStudies in animals have documented that, compared with glucose, dietary fructose induces dyslipidemia and insulin resistance. To assess the relative effects of these dietary sugars during sustained consumption in humans, overweight and obese subjects consumed glucose- or fructose-sweetened beverages providing 25% of energy requirements for 10 weeks. Although both groups exhibited similar weight gain during the intervention, visceral adipose volume was significantly increased only in subjects consuming fructose. Fasting plasma triglyceride concentrations increased by approximately 10% during 10 weeks of glucose consumption but not after fructose consumption. In contrast, hepatic de novo lipogenesis (DNL) and the 23-hour postprandial triglyceride AUC were increased specifically during fructose consumption. Similarly, markers of altered lipid metabolism and lipoprotein remodeling, including fasting apoB, LDL, small dense LDL, oxidized LDL, and postprandial concentrations of remnant-like particle-triglyceride and -cholesterol significantly increased during fructose but not glucose consumption. In addition, fasting plasma glucose and insulin levels increased and insulin sensitivity decreased in subjects consuming fructose but not in those consuming glucose. These data suggest that dietary fructose specifically increases DNL, promotes dyslipidemia, decreases insulin sensitivity, and increases visceral adiposity in overweight/obese adults.

    www.ncbi.nlm.nih.gov/...PMC2673878 fructose glucose nutrition health sweetner

  • Jan 24, 10

    www.steadyhealth.com/...ur_Brain_Function_a794_f0.html health food brain

    • brain uses carbohydrates for energy and omega-3 fatty acids for the formation of its cell structure.
    • omega-3 fatty acids

    46 more annotations...

  • May 11, 10

    Research reveals that children who eat lunch as part of the National School Lunch Program (NSLP) have an increased likelihood of becoming overweight...The findings also show that the School Breakfast Program (SBP) is a "valuable tool in the current battle against childhood obesity," according to the research.

    www.sciencedaily.com/...100511092147.htm no tag

    • The findings also show that the School Breakfast Program (SBP) is a "valuable tool in the current battle against childhood obesity," according to the research.
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