This link has been bookmarked by 4 people . It was first bookmarked on 25 Jul 2008, by Sue Cifelli.
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10 Mar 17
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Detected calcium arterial deposits thus permit life style changes to be instituted before sudden death or acute myocardial infarction has occurred. This increased risk of calcium deposition into arteries has recently been confirmed to bring increased risk of heart attack and heart disease deaths to blacks, Hispanics and Chinese[1] even though their risks are less than Caucasians.
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high protein, high dairy, high phosphorus acidifying diet. This type food causes large amounts of calcium to be wasted in the urine as it is removed from bone tissue to try to preserve an alkaline cellular environment in the face of a very acidic dietary protein intake.
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The nation of Thailand which eats almost no dairy products and obtains calcium primarily from vegetables has much less osteoporosis than western nations on their high protein high dairy product diets.
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At age 80 the average calcium content in the aorta is 140 times greater[2] than the levels of aortic calcification noted at age 40. This may relate to a long period of unrecognized Vitamin K2 deficiency.
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Vitamin K2 is found in animals and bacteria (healthy colon bacteria, Japanese natto,
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Studies have shown that subclinical Vitamin K deficiency,[3] [4] is present in most healthy adults. The first symptoms of this deficiency can be heart attack or a fractured osteoporotic bone.
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Vitamin K1 was less effective than Vitamin K2 in preventing bone loss.
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K2 to levels 7-8 fold higher than that seen after one dose. Vitamin K2(MK-7) is 6 times more potent than Vitamin K1.
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Use Of Vitamin K2(Menaquinone-7) To Prevent Calcium Plaques From Appearing In Arteries
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A clinical study from Rotterdam, Holland revealed a correlation between long term adequate Vitamin K2 intake and a lower incidence of calcification of the wall of the aorta. Arteries with no plaques have a 20 to 50 fold increase in Vitamin K2
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concentration when compared to arteries with arterial plaques
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The high K2(menaquinone-7) content arteries were noted to be more flexible[7] and elastic than arteries lacking K2.
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A protein called osteocalcin transports calcium to bone. Vitamin K2(menaquinone-7) is used to solidify this calcium into the bone matrix. When Vitamin K2 is lacking the calcium remains in the blood and ends up getting deposited in the walls of arteries and other sites which is very undesirable.
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Vitamin K2 becomes a critical nutrient for both bone and arteries. The primary therapy for osteoporosis in Japan has become Vitamin K2(Synergy K).
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They learned that persons with the highest dietary intake of K2 (primarily originating in low fat Dutch cheeses Gouda and Edam) had the least evidence of calcification of the aorta[8] when compared to persons with low Vitamin K2 intakes. The higher the intake of these cheeses the lower the mortality from cardiovascular disease.
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All of the Vitamin K2 produced in making the enzyme nattokinase has now become available to be sold for use in food supplements
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The calcium[10] content of arteries is now proven to be more dangerous than diabetes, elevated cholesterol or hypertension, we must now try to educate patients.
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Vitamin K2 is a critical nutrient for patients with arteriosclerosis as it has the potential to prevent and remove calcium from arteriosclerotic plaques thus making plaques easier to dissolve and less dangerous..
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Methionine from red meat, milk, and milk products is converted in the body into homocysteine. When the body’s stores of B6 (pyridoxine), folic acid, and B12 fail to bring this homocysteine down to normal values
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Homocysteine stops the production of the valuable vasodilating nitric acid, causes blood to thicken, and facilitates the oxidation of LDL cholesterol, thus setting the stage for an atherosclerotic plaque to form. As more patients are studied, it has become evident that elevated levels of homocysteine are a common cause for arteriosclerosis (at least 40% of patients).
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A Norwegian[11] study discovered that in 587 patients with coronary heart disease, the risk of death within four years was proportional to total plasma homocysteine level. The risk rose from 3.8% with homocysteine below nine micromols per liter to 24.7% in patients with homocysteine levels above 15 micromols per liter.
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Research by Dr. Linus Pauling and Dr. Mattias Rath demonstrated that 2 grams of Vitamin C and 2 grams of the amino acid Lysine each taken three times daily was very effective in healing arteriosclerotic arteries.
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These lipoprotein deposits are transported to the liver where they are burned up. Thus the size of the plaque starts to decrease. This is a natural process which proceeds uneventfully until the plaque is gone.
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Lipoprotein(a) is a 10 times more dangerous risk factor than Low Density Lipoprotein or cholesterol. Within 6 to 8 weeks of starting lysine and vitamin C many patients had experienced loss of anginal pain, disappearance of hypertension and the ability to pass an exercise treadmill test
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Patients lacking Vitamin C use the sticky substance lipoprotein (a) to try to solidify the weak spots in arteries which lack collagen fibers. Lipoprotein (a) sticks to irregular areas on the lining of the arteries and proceeds to collect platelets, calcium, lipoproteins (LDL), lipoprotein (a) and fibrin from the arterial blood flow. Over time scar tissue appears and smooth muscle fibers may appear. This build up of the plaque reduces the speed of blood flow and interferes with proper oxygenation of tissues.
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Pathologists have long observed that arterial plaques are commonly seen where the forceful arterial stream strikes an arterial wall. This location is frequently at the site where the coronary arteries branch off the aorta. The steady strong force of the arterial stream causes the weak artery wall lacking Vitamin C to seek the reinforcement of additional lipoprotein (a) making a buildup of large plaques at this site very common.
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If cholesterol was a dangerous toxic substance the occurrence of arterial plaques would be completely random rather than appearing at sites where the arterial wall is being steadily traumatized. This information creates a strong argument that cholesterol is not a primary cause for vascular disease. Additionally we know that more than 50% of patients having an acute myocardial infraction have perfectly normal cholesterol values.
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Lipoprotein (a) values can be lowered by 36% in patients taking 2 to 4 grams of Vitamin B3 (nicotinic acid).
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Vitamin C alone or combined with nicotinic acid may have a lowering effect on the production of lipoproteins .resulting in lower lipoprotein values. When these therapies are combined with lysine and proline the cardiovascular risk of lipoprotein (a) can be reduced.
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nimals that easily produce Vitamin C have low levels of lipoprotein (a). Thus humans, guinea pigs and a few primates that fail to produce Vitamin C are able to inadequately repair arteries by virtue of having access to lipoprotein (a).
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ascorbate
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25 Jul 08
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